понедельник, 3 ноября 2008 г.

Caisson disease.

The clinic, Caisson disease develops owing to penetration of nitrogen of tissues into blood at transition from under the raised pressure (2-5 ) to the normal. Passing from tissues in vascular system, nitrogen causes a gas vascular embolism. Arising emboluses, merging, can have foamy structure. In other cases gas blisters frame conditions for ultram 100mg formation of an occlusive thrombus from formulated elements of blood. Thus, the caisson disease proceeds or in the form of an aeroembolism, or an aerothrombosis.
Illness can be acute and chronic. Acute forms of a caisson disease are caused by an aeroembolism, chronic - an aerothrombosis. Chronic forms can be primary and secondary. Initially-chronic forms arise slowly, pass the long latent period and are caused by an aerothrombosis. The osteoarthrosis and a myocardial dystrophy concern them. Again-chronic forms are caused by an aeroembolism before which elimination in a nervous tissue there were deep irreversible pathological changes.
There is a certain dependence of gravity of a current of symptoms on the sizes and quantity of gas emboluses. In this connection among acute forms of a caisson disease it is necessary to distinguish easy, moderately severe, serious and lethal. Easy forms are characterised by convertibility after elimination of blockade of a circulation and influence of emboluses. It is necessary to carry the aeroembolisms which elimination is accompanied by the insignificant residual phenomena to moderately severe forms. Serious forms develop at insufficiently timely elimination of embolic action of gas or at massive aeroembolisms. They come to an end with the expressed defect of functions of organs. In case of massive flooding by aeroemboluses serious forms of a caisson disease can become lethal. Lethal forms of a caisson disease are observed at full blockade of a pulmonary circulation, acute insufficiency of work of heart or circulation disturbance in an oblong brain.
From the moment of the termination of decompression (transition from under the raised pressure to normal) and before occurrence of the first symptoms of a caisson disease passes usually not less a half an hour. Than above pressure and degree of a saturation of an organism (saturation by gas) and the more shortly decompression, the will come a gas embolism more likely. If saturation degree is low, for development of a gas embolism more long time is necessary, and disease will be expressed in easier form. vpxl 200mg
Osteoarthralgias concern easy forms, a neuralgia and dermal lesions (an itch, and a skin emphysema).
The basic symptom of an osteoarthralgia is the pain which is localised in bones and joints of extremities. Quite often pains are preceded by paresthesias. Intensity of pains can have alternating character. Sometimes pains are accompanied by cramps and fibrillar twitchings in extremities, a tumescence with exudate presence in joints. At a palpation the crepitation is taped. Rise in temperature can be observed.

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